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Lncrna Malat1 And Mir-26 Cooperate In The Regulation Of

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lncRNA Malat1 and miR-26 cooperate in the regulation of neuronal progenitor cell proliferation and differentiation Supplemental Material Supplemental_Figure_Legends.docx

The lncRNA Malat1 regulates microvascular function after

The study explores the role of lncRNAs in hepatocellular carcinoma (HCC) and elucidates the specific molecular mechanisms by which key lncRNAs such as HULC and

lncRNA Malat1 and miR-26 cooperate in the regulation of neuronal ...

Long noncoding RNAs (lncRNAs) are effective regulators of both RNA and protein functions throughout cell biology, including viral replication. Emerging studies have shown that lncRNAs

As a member of the lncRNA family, MALAT1 is a multi-functional lncRNA and an important regulator in hepatic fibrosis, liver regeneration, cancer, and fatty liver diseases. The

  • MALAT1 long non-coding RNA in cancer
  • LncRNA-MALAT1: A Key Participant in the Occurrence and
  • The lncRNA Malat1 regulates microvascular function after
  • Potential roles of lncRNA MALAT1-miRNA interactions in

Objectives This study aimed to investigate the regulatory roles of lncRNA MALAT1, miR-124-3p, and IGF2BP1 in osteogenic differentiation of periodontal ligament stem

In a study, the expression of MALAT1 in rat OP models is down regulated. However, it is also shown that MALAT1 can induce inhibition of osteogenic differentiation by

Our present study reported that silencing Malat1 exacerbated microvascular dysfunction by enhancing CMEC apoptosis partly through regulation of miR-26b-5p/Mfn1

Cancer hallmarks regulated by MALAT1. (A) Regulation of growth and invasion by MALAT1. Histone demethylase JMJD2C binds and activates MALAT1 promoter leading to the

MALAT1 can promote the expression of NLR family pyrin domain containing 3 (NLRP3) by down-regulating the miR-224-5p level by acting as a competitive endogenous RNA. It has been

Scientific Reports – LncRNA NEAT1 regulation of the miR-101-3p/RAC1 axis affects cervical cancer aerobic glycolysis and progression Skip to main content Thank you for visiting

有趣的是，这与分化的 ESC 中 miR-26 家族成员 miR-26a 和 miR-26b 表达的增加相一致。 miR-26a/b 失活可挽救Malat1敲除 (KO) 细胞的增殖表型，并导致复合 Malat1KO/mir

In spite of its high mortality rate and difficulty in finding a cure, scientific advancements have contributed to a reduction in cancer-related fatalities. Aberrant gene

lncRNA Malat1 and miR-26 cooperate in the regulation of neuronal progenitor cell proliferation and differentiation. 被引：4 | 作者 ： Was, Nina [1 ]

Abstract. Background/Aims: LncRNA metastasis associated lung adenocarcinoma transcript 1 (MALAT1) was reported to be highly expressed in an in vitro mimic of ischemic

Malat1 gene is located within human chromosome 11q13 and mouse chromosome 19qA. 10,23 It is abundantly expressed at a level comparable with or even higher than many protein-coding

  • Roles of long noncoding RNAs in human inflammatory diseases
  • The role of a ceRNA regulatory network based on lncRNA
  • lncRNA Malat1和miR-26协同调节神经祖细胞增殖和分化,RNA
  • LncRNA MALAT1 facilitates HIV-1 replication by upregulation of

The regulation of MALAT1 transcripts by associated proteins has also been demonstrated for TDP-43. TDP-43 was originally identified as a DNA-binding protein that binds

LncRNA MALAT1/miR-181a-5p affects the proliferation and adhesion of myeloma cells via regulation of Hippo-YAP signaling pathway . Yanbei Sun a Department of Nephrology,

Silencing of lncRNA MALAT1 increases miR-558 to promote viability and ...

In order to further reveal the roles of MALAT1 in gene regulation, we constructed a MALAT1-TFs-Targets network by mapping the TFs with changes at the protein level and the

Studies have shown that long-chain non-coding RNA (LncRNA) improves DN by regulating microRNA (miR). The study intended to investigate the role of MALAT1/miR-206 on

Here we analyzed the function of the long non-coding (lnc)RNA Malat1 during neural embryonic stem cell (ESC) differentiation. We find that deletion of Malat1 leads to

We find that deletion of Malat1 leads to inhibition of proliferation of neural progenitor cells (NPCs). Interestingly, this coincides with an increase in the expression of miR-26 family members miR

Long non-coding RNAs (lncRNAs) are non-protein coding transcripts that are longer than 200 nucleotides in length. LncRNAs are implicated in gene expression at the

Neurogenesis is a finely tuned process, which depends on the balanced execution of expression programs that regulate cellular differentiation and proliferation. Different

Overexpression of the lncRNA MALAT1 could promote the production of IL-6, IL-8, and TNF-α through regulation of the miR-590/STAT3 LDL and LOX-1 in atherogenesis. Curr

The positive correlation between MALAT1 and foxq1 expression as well as the inhibitory effect of MALAT1 on miR-124 expression indicate that MALAT1 can regulates this

We find that deletion of Malat1 leads to inhibition of proliferation of neural progenitor cells (NPCs). Interestingly, this coincides with an increase in the expression of miR-26 family members miR

The present study elucidated the functions and mechanisms of Malat1 in cardiac microcirculation repair after MI. The underlying mechanisms of the effects of Malat1 could be attributed to its

Overall, the collected evidence from various studies suggests that MALAT1 interacts with and regulates the expression of multiple miRNAs, including miR-124, miR-205,